Response to: Correspondence on 'H3.3K27M mutation is not a suitable target for immunotherapy in HLA-A2+ patients with diffuse midline glioma' by Chheda<i>et al</i>-Reference-Cited by-同舟云学术

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Response to: Correspondence on 'H3.3K27M mutation is not a suitable target for immunotherapy in HLA-A2+ patients with diffuse midline glioma' by Chhedaet al

Published:2023-03 Issue:3 Volume:11 Page:e006784
ISSN:2051-1426
Container-title:Journal for ImmunoTherapy of Cancer
language:en
Short-container-title:J Immunother Cancer

Author:

Immisch Lena^ORCID,Papafotiou George,Popp Oliver,Mertins Philipp,Blankenstein Thomas,Willimsky Gerald^ORCID

Funder

Deutsche Krebshilfe

Helmholtz-Gemeinschaft, Zukunftsthema ‘Immunology and Inflammation’

DKTK joint funding

European Union Agency for Network and Information Security

German Research Foundation

Berlin Institute of Health

Publisher

BMJ

Subject

Cancer Research,Pharmacology,Oncology,Molecular Medicine,Immunology,Immunology and Allergy

Reference8 articles.

1. Chen K-Y , Bush K , Klein RH , et al . Reciprocal H3.3 gene editing identifies K27M and G34R mechanisms in pediatric glioma including Notch signaling. Commun Biol 2020;3:363. doi:10.1038/s42003-020-1076-0

2. H3.3K27M mutation is not a suitable target for immunotherapy in HLA-A2+ patients with diffuse midline glioma;Immisch;J Immunother Cancer,2022

3. Novel and shared neoantigen derived from histone 3 variant H3.3K27M mutation for glioma T cell therapy

4. Lowe DJ , Herzog M , Mosler T , et al . Chronic irradiation of human cells reduces histone levels and deregulates gene expression. Sci Rep 2020;10:2200. doi:10.1038/s41598-020-59163-4

5. Ionizing radiation affects 26s proteasome function and associated molecular responses, even at low doses

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