Helicobacter pylori associated phospholipase A2 activity: a factor in peptic ulcer production?

Author:

Langton S R,Cesareo S D

Publisher

BMJ

Subject

General Medicine,Pathology and Forensic Medicine

Reference30 articles.

1. Gastric Campylobacter-like organisms, gastritis, and peptic ulcer disease;Blaser, M.J.;Gastroenterology,1987

2. Cytotoxic activity in broth-culture filtrates of Campylobacter pylori;J Med Microbiol,1988

3. Cytotoxin production by Campylobacter pylori strains isolated from patients with peptic ulcers and from patients with chronic gastritis only;Figura, N.; Guglielmetti, P.; Rossolini, A.;J Clin Microbiol,1989

4. Hypothesis: Helicobacter pylori, urease, mucus, and gastric ulcer;Sidebottom, R.L.; Baron, J.H.;Lancet,1990

5. Campylobacter pyloridis and acid induced gastric metaplasia in the pathogenesis of duodenitis;Wyatt, J.I.; Rathbone, B.J.; Dixon, M.F.; Headley, R.V.;J Clin Pathol,1987

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