Oxidative polymorphism of debrisoquine in Parkinson's disease.

Author:

Benitez J,Ladero J M,Jimenez-Jimenez F J,Martinez C,Puerto A M,Valdivielso M J,Llerena A,Cobaleda J,Munoz J J

Publisher

BMJ

Subject

Psychiatry and Mental health,Neurology (clinical),Surgery

Reference18 articles.

1. Polymorphic hydroxylation of debrisoquine in man;Mahgoub, A.; Idle, JR, Dring; LG, Lancaster; R, Smith; R.L.;Lancet,1977

2. Extensive metabolizers of debrisoquine become poor metabolizers during quinidine treatment;Brosen, K.; Gram, L.F.; Haghfelt, T.; Bertilsson, L.;Pharmacol Toxicol,1987

3. Aetiology of Parkinson's disease;Calne, D.B.; Langston, J.W.;Lancet,1983

4. A family and population study of the genetic polymorphism of debrisoquine in a white British population;Evans, D.A.P.; Mahgoub, A.; Sloan, T.P.; Idle, JR, Smith; R.L.;J Med Genet,1980

5. Metabolism of the neurotoxic tertiary amine, MPTP, by brain monoamine oxidase;Chiba, K.; Trevor, A.; Castagnoli, N.;Biochem Biophys Res Commun,1984

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