Diurnal variation of NMDA receptor expression in the rat cerebral cortex is associated with traumatic brain injury damage
Author:
Funder
CONACYT
PAPIIT
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
http://link.springer.com/content/pdf/10.1186/s13104-018-3258-0.pdf
Reference52 articles.
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2. Armstead WV. Age dependent NMDA contribution to impaired hypotensive cerebral hemodynamics following brain injury. Brain Res Dev Brain Res. 2002;139:19–28.
3. Okiyama K, Smith DH, White WF, Richter K, McIntosh TK. Effects of the novel NMDA antagonists CP-98,113, CP-101,581 and CP-101,606 on cognitive function and regional cerebral edema following experimental brain injury in the rat. J Neurotrauma. 1997;14:211–22.
4. Bauer R, Walter B, Fritz H, Zwiener U. Ontogenetic aspects of traumatic brain edema-facts and suggestions. Exp Toxicol Pathol. 1999;51:143–50.
5. Obrenovitch TP, Urenjak J. Is high extracellular glutamate the key to excitotoxicity in traumatic brain injury? J Neurotrauma. 1997;14:677–98.
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