Biological network inferences for a protection mechanism against familial Creutzfeldt-Jakob disease with E200K pathogenic mutation-Reference-Cited by-同舟云学术

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Biological network inferences for a protection mechanism against familial Creutzfeldt-Jakob disease with E200K pathogenic mutation

Published:2014-08-22 Issue:1 Volume:7 Page:
ISSN:1755-8794
Container-title:BMC Medical Genomics
language:en
Short-container-title:BMC Med Genomics

Author:

Lee Sol Moe,Chung Myungguen,Hwang Kyu Jam,Ju Young Ran,Hyeon Jae Wook,Park Jun-Sun,Kim Chi-Kyeong,Choi Sangho,Lee Jeongmin,Kim Su Yeon

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics

Link

http://link.springer.com/content/pdf/10.1186/1755-8794-7-52.pdf

Reference53 articles.

1. Kitamoto T, Muramoto T, Mohri S, Doh-Ura K, Tateishi J: Abnormal isoform of prion protein accumulates in follicular dendritic cells in mice with Creutzfeldt-Jakob disease. J Virol. 1991, 65 (11): 6292-6295.

2. Taraboulos A, Serban D, Prusiner SB: Scrapie prion proteins accumulate in the cytoplasm of persistently infected cultured cells. J Cell Biol. 1990, 110 (6): 2117-2132.

3. Tateishi J, Kitamoto T: Inherited prion diseases and transmission to rodents. Brain Pathol. 1995, 5 (1): 53-59.

4. Collins S, McLean CA, Masters CL: Gerstmann-Straussler-Scheinker syndrome, fatal familial insomnia, and kuru: a review of these less common human transmissible spongiform encephalopathies. J Clin Neurosci. 2001, 8 (5): 387-397.

5. Tateishi J, Kitamoto T, Hoque MZ, Furukawa H: Experimental transmission of Creutzfeldt-Jakob disease and related diseases to rodents. Neurology. 1996, 46 (2): 532-537.

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