Cu(II) enhances the effect of Alzheimer’s amyloid-β peptide on microglial activation-Reference-Cited by-同舟云学术

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Cu(II) enhances the effect of Alzheimer’s amyloid-β peptide on microglial activation

Published:2015-06-24 Issue:1 Volume:12 Page:
ISSN:1742-2094
Container-title:Journal of Neuroinflammation
language:en
Short-container-title:J Neuroinflammation

Author:

Yu Fengxiang,Gong Ping,Hu Zhuqin,Qiu Yu,Cui Yongyao,Gao Xiaoling,Chen Hongzhuan,Li Juan

Funder

National Natural Science Foundation of China

International Science & Technology Cooperation Program of China

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Immunology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1186/s12974-015-0343-3.pdf

Reference46 articles.

1. Niranjan R. Molecular basis of etiological implications in Alzheimer’s disease: focus on neuroinflammation. Mol Neurobiol. 2013;48:412–28.

2. Bachstetter AD, Norris CM, Sompol P, Wilcock DM, Goulding D, Neltner JH, et al. Early stage drug treatment that normalizes proinflammatory cytokine production attenuates synaptic dysfunction in a mouse model that exhibits age-dependent progression of Alzheimer’s disease-related pathology. J Neurosci. 2012;32:10201–10.

3. Karran E, Mercken M, De Strooper B. The amyloid cascade hypothesis for Alzheimer’s disease: an appraisal for the development of therapeutics. Nat Rev Drug Discov. 2011;10:698–712.

4. Li Y, Tan MS, Jiang T, Tan L. Microglia in Alzheimer’s disease. Biomed Res Int. 2014;2014:437483.

5. Jekabsone A, Mander PK, Tickler A, Sharpe M, Brown GC. Fibrillar beta-amyloid peptide Aβ1-40 activates microglial proliferation via stimulating TNF-α release and H2O2 derived from NADPH oxidase: a cell culture study. J Neuroinflammation. 2006;3:24.

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