STAT3 activation is required for the antiapoptotic effects of prolactin in cervical cancer cells-Reference-Cited by-同舟云学术

STAT3 activation is required for the antiapoptotic effects of prolactin in cervical cancer cells

Published:2015-09-04 Issue:1 Volume:15 Page:
ISSN:1475-2867
Container-title:Cancer Cell International
language:en
Short-container-title:Cancer Cell Int

Author:

de Arellano Adrián Ramírez,Lopez-Pulido Edgar I.,Martínez-Neri Priscila A.,Chávez Ciro Estrada,Lucano Renee González,Fafutis-Morris Mary,Aguilar-Lemarroy A.,Muñoz-Valle José. F.,Pereira-Suárez Ana Laura

Funder

SEP CONACYT 2013

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Oncology

Link

http://link.springer.com/content/pdf/10.1186/s12935-015-0234-9

Reference33 articles.

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2. Hu ZZ, Meng J, Dufau ML (2001) Isolation and characterization of two novel forms of the human prolactin receptor generated by alternative splicing of a newly identified exon 11. J Biol Chem 276(44):41086–41094

3. Kline JB, Roehrs H, Clevenger CV (1999) Functional characterization of the intermediate isoform of the human prolactin receptor. J Biol Chem 274(50):35461–35468

4. Trott JF, Hovey RC, Koduri S, Vonderhaar BK (2003) Alternative splicing to exon 11 of human prolactin receptor gene results in multiple isoforms including a secreted prolactin-binding protein. J Mol Endocrinol 30(1):31–47

5. Welsch CW, Nagasawa H (1977) Prolactin and murine mammary tumorigenesis: a review. Cancer Res 37(4):951–963

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