Endothelial glycocalyx degradation is more severe in patients with non-pulmonary sepsis compared to pulmonary sepsis and associates with risk of ARDS and other organ dysfunction-Reference-Cited by-同舟云学术

Endothelial glycocalyx degradation is more severe in patients with non-pulmonary sepsis compared to pulmonary sepsis and associates with risk of ARDS and other organ dysfunction

Published:2017-10-06 Issue:1 Volume:7 Page:
ISSN:2110-5820
Container-title:Annals of Intensive Care
language:en
Short-container-title:Ann. Intensive Care

Author:

Murphy Laura S.,Wickersham Nancy,McNeil J. Brennan,Shaver Ciara M.,May Addison K.,Bastarache Julie A.,Ware Lorraine B.

Funder

National Institutes of Health

Vanderbilt University School of Medicine

Publisher

Springer Science and Business Media LLC

Subject

Critical Care and Intensive Care Medicine

Link

http://link.springer.com/content/pdf/10.1186/s13613-017-0325-y.pdf

Reference49 articles.

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2. Schmidt EP, Lee WL, Zemans RL, Yamashita C, Downey GP. On, around, and through: neutrophil-endothelial interactions in innate immunity. Physiol (Bethesda, Md.). 2011;26(5):334–47.

3. Curry FE, Adamson RH. Endothelial glycocalyx: permeability barrier and mechanosensor. Ann Biomed Eng. 2012;40(4):828–39.

4. Florian JA, Kosky JR, Ainslie K, Pang Z, Dull RO, Tarbell JM. Heparan sulfate proteoglycan is a mechanosensor on endothelial cells. Circ Res. 2003;93(10):e136–42.

5. Grundmann S, Fink K, Rabadzhieva L, et al. Perturbation of the endothelial glycocalyx in post cardiac arrest syndrome. Resuscitation. 2012;83(6):715–20.

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