Upregulation of CKIP-1 inhibits high-glucose induced inflammation and oxidative stress in HRECs and attenuates diabetic retinopathy by modulating Nrf2/ARE signaling pathway: an in vitro study-Reference-Cited by-同舟云学术

Upregulation of CKIP-1 inhibits high-glucose induced inflammation and oxidative stress in HRECs and attenuates diabetic retinopathy by modulating Nrf2/ARE signaling pathway: an in vitro study

Published:2019-08-23 Issue:1 Volume:9 Page:
ISSN:2045-3701
Container-title:Cell & Bioscience
language:en
Short-container-title:Cell Biosci

Author:

Zhang Lan,Yu Jie,Ye Mingxia,Zhao Hailan

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13578-019-0331-x.pdf

Reference39 articles.

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2. Ung C, Sanchez AV, Shen L, Davoudi S, Ahmadi T, Navarro-Gomez D, et al. Whole exome sequencing identification of novel candidate genes in patients with proliferative diabetic retinopathy. Vision Res. 2017;139:168–76.

3. Zhang ZZ, Qin XH, Zhang J. MicroRNA-183 inhibition exerts suppressive effects on diabetic retinopathy by inactivating BTG1-mediated PI3K/Akt/VEGF signaling pathway. Am J Physiol Endocrinol Metab. 2019;316:E1050–60.

4. Shao Y, Dong LJ, Takahashi Y, Chen J, Liu X, Chen Q, et al. miRNA-451a regulates RPE function through promoting mitochondrial function in proliferative diabetic retinopathy. Am J Physiol Endocrinol Metab. 2019;316(3):E443–52.

5. Calderon GD, Juarez OH, Hernandez GE, Punzo SM, De la Cruz ZD. Oxidative stress and diabetic retinopathy: development and treatment. Eye (London, England). 2017;31(8):1122–30.

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