Neuroinflammation in Parkinson’s disease and its potential as therapeutic target-Reference-Cited by-同舟云学术

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Neuroinflammation in Parkinson’s disease and its potential as therapeutic target

Published:2015-10-12 Issue:1 Volume:4 Page:
ISSN:2047-9158
Container-title:Translational Neurodegeneration
language:en
Short-container-title:Transl Neurodegener

Author:

Wang Qinqin,Liu Yingjun,Zhou Jiawei

Funder

National Natural Science Foundation of China (CN)

Ministry of Science and Technology of the People's Republic of China (CN)

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Cognitive Neuroscience,Clinical Neurology

Link

http://link.springer.com/content/pdf/10.1186/s40035-015-0042-0

Reference125 articles.

1. Fahn S. Description of Parkinson’s disease as a clinical syndrome. Ann N Y Acad Sci. 2003;991:1–14.

2. von Campenhausen S, Bornschein B, Wick R, Botzel K, Sampaio C, Poewe W, et al. Prevalence and incidence of Parkinson’s disease in Europe. Eur Neuropsychopharmacol. 2005;15:473–90.

3. Wirdefeldt K, Adami HO, Cole P, Trichopoulos D, Mandel J. Epidemiology and etiology of Parkinson’s disease: a review of the evidence. Eur J Epidemiol. 2011;26 Suppl 1:S1–58.

4. Waak J, Weber SS, Waldenmaier A, Gorner K, Alunni-Fabbroni M, Schell H, et al. Regulation of astrocyte inflammatory responses by the Parkinson’s disease-associated gene DJ-1. FASEB J. 2009;23:2478–89.

5. Mori F, Piao YS, Hayashi S, Fujiwara H, Hasegawa M, Yoshimoto M, et al. Alpha-synuclein accumulates in Purkinje cells in Lewy body disease but not in multiple system atrophy. J Neuropathol Exp Neurol. 2003;62:812–9.

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