Reduced presynaptic vesicle stores mediate cellular and network plasticity defects in an early-stage mouse model of Alzheimer’s disease-Reference-Cited by-同舟云学术

Reduced presynaptic vesicle stores mediate cellular and network plasticity defects in an early-stage mouse model of Alzheimer’s disease

Published:2019-01-22 Issue:1 Volume:14 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Chakroborty Shreaya,Hill Evan S.,Christian Daniel T.,Helfrich Rosalind,Riley Shannon,Schneider Corinne,Kapecki Nicolas,Mustaly-Kalimi Sarah,Seiler Figen A.,Peterson Daniel A.,West Anthony R.,Vertel Barbara M.,Frost William N.,Stutzmann Grace E.^ORCID

Funder

National Institute on Aging

Rosalind Franklin University of Medicine and Science

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13024-019-0307-7.pdf

Reference120 articles.

1. Briggs CA, Chakroborty S, Stutzmann GE. Emerging pathways driving early synaptic pathology in Alzheimer's disease. Biochem Biophys Res Commun. 2017;483(4):988–97 Epub 2016/09/24.

2. Chakroborty S, Stutzmann GE. Early calcium dysregulation in Alzheimer’s disease: setting the stage for synaptic dysfunction. Sci China Life Sci. 2011;54(8):752–62.