Ceftriaxone Modulates Ubiquitination of α-Amino-3-Hydroxy-5-Methyl-4-Isoxazole Propionic Acid Receptors to Improve Long-Term Potentiation Impairment Induced by Exogenous β-Amyloid in a Glutamate Transporter-1 Dependent Manner-Reference-Cited by-同舟云学术

Ceftriaxone Modulates Ubiquitination of α-Amino-3-Hydroxy-5-Methyl-4-Isoxazole Propionic Acid Receptors to Improve Long-Term Potentiation Impairment Induced by Exogenous β-Amyloid in a Glutamate Transporter-1 Dependent Manner

Published:2024-02-19 Issue:9 Volume:61 Page:7181-7194
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Jiang Rui,Li Li,Wang Bu,Liu Lizhe,Liu Lirong,Xian Xiaohui,Li Wenbin

Funder

National Natural Science Foundation of China

Natural Science Foundation of Hebei Province and Precision Medicine Joint Fund

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1007/s12035-024-04037-3.pdf

Reference45 articles.

1. Mattson MP (2004) Pathways towards and away from Alzheimer’s disease. Nature 430:631–639. https://doi.org/10.1038/nature02621

2. Hardy J, Selkoe DJ (2002) The amyloid hypothesis of Alzheimer’s disease: progress and problems on the road to therapeutics. Science 297:353–356. https://doi.org/10.1126/science.1072994

3. Lacor PN, Buniel MC, Furlow PW et al (2007) Abeta oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer’s disease. J Neurosci 27:796–807. https://doi.org/10.1523/JNEUROSCI.3501-06.2007

4. Nalivaeva NN, Belyaev ND, Kerridge C et al (2014) Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer’s disease. Front Aging Neurosci 6:235. https://doi.org/10.3389/fnagi.2014.00235

5. Ramirez S (2018) Crystallizing a memory. Science 360:1182–1183. https://doi.org/10.1126/science.aau0043