Increased cerebrospinal fluid soluble TREM2 concentration in Alzheimer’s disease-Reference-Cited by-同舟云学术

Increased cerebrospinal fluid soluble TREM2 concentration in Alzheimer’s disease

Published:2016-01-12 Issue:1 Volume:11 Page:
ISSN:1750-1326
Container-title:Molecular Neurodegeneration
language:en
Short-container-title:Mol Neurodegeneration

Author:

Heslegrave Amanda,Heywood Wendy,Paterson Ross,Magdalinou Nadia,Svensson Johan,Johansson Per,Öhrfelt Annika,Blennow Kaj,Hardy John,Schott Jonathan,Mills Kevin,Zetterberg Henrik

Funder

Wolfson Foundation

Alzheimer’s Research UK

Svenska Forskningsrådet Formas

Biomedical Research Council (GOSH)

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Molecular Biology

Link

http://link.springer.com/content/pdf/10.1186/s13024-016-0071-x

Reference37 articles.

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3. Klunemann HH, Ridha BH, Magy L, Wherrett JR, Hemelsoet DM, Keen RW, et al. The genetic causes of basal ganglia calcification, dementia, and bone cysts: DAP12 and TREM2. Neurology. 2005;64(9):1502–7.

4. Guerreiro RJ, Lohmann E, Bras JM, Gibbs JR, Rohrer JD, Gurunlian N, et al. Using exome sequencing to reveal mutations in TREM2 presenting as a frontotemporal dementia-like syndrome without bone involvement. JAMA neurology. 2013;70(1):78–84.

5. Guerreiro R, Wojtas A, Bras J, Carrasquillo M, Rogaeva E, Majounie E, et al. TREM2 variants in Alzheimer’s disease. N Engl J Med. 2013;368(2):117–27.

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