Monoamine oxidase-B (MAO-B) inhibitors: implications for disease-modification in Parkinson’s disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Cognitive Neuroscience,Clinical Neurology
Link
http://link.springer.com/content/pdf/10.1186/2047-9158-2-19.pdf
Reference62 articles.
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2. Braak H, Del Tredici K, Rüb U, et al.: Staging of brain pathology related to sporadic Parkinson's disease. Neurobiol Aging 2003, 24: 197-211. 10.1016/S0197-4580(02)00065-9
3. Riederer P, Youdim MB: Monoamine oxidase activity and monoamine metabolism in brains of parkinsonian patients treated with l-deprenyl. J Neurochemistry 1986, 46: 1359-1365. 10.1111/j.1471-4159.1986.tb01747.x
4. Cohen G, Pasik P, Cohen B, et al.: Pargyline and deprenyl prevent the neurotoxicity of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) in monkeys. Eur J Pharmacology 1984, 106: 209-210. 10.1016/0014-2999(84)90700-3
5. Finnegan KT, Skratt JJ, Irwin I, et al.: Protection against DSP-4-induced neurotoxicity by deprenyl is not related to its inhibition of MAO B. Eur J Pharmacol 1990, 184: 119-126. 10.1016/0014-2999(90)90672-S
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