Inflachromene ameliorates Parkinson's disease by targeting Nrf2-binding Keap1-Reference-Cited by-同舟云学术

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Inflachromene ameliorates Parkinson's disease by targeting Nrf2-binding Keap1

Published:2024 Issue:10 Volume:15 Page:3588-3595
ISSN:2041-6520
Container-title:Chemical Science
language:en
Short-container-title:Chem. Sci.

Author:

Yim Junhyeong¹^ORCID,Hwang Yoon Soo²^ORCID,Lee Jae-Jin³,Kim Ju Hee³,Baek Jeong Yeob⁴^ORCID,Jeong Jaeyeong⁴,Choi Young Il³^ORCID,Jin Byung Kwan⁴,Park Seung Bum¹²³^ORCID

Affiliation:

1. Department of Biophysics and Chemical Biology, Seoul National University, Seoul 08826, Korea

2. CRI Center for Chemical Proteomics, Department of Chemistry, Seoul National University, Seoul 08826, Korea

3. SPARK Biopharma, Inc., Seoul, 08791, Korea

4. Department of Biochemistry & Molecular Biology, School of Medicine, Kyung Hee University, Seoul 02447, Korea

Abstract

Target identification using fluorescence difference in two-dimensional gel electrophoresis (FITGE) revealed the molecular mechanism of ICM that ameliorates PD by targeting a specific form of Keap1.

Publisher

Royal Society of Chemistry (RSC)

Link

http://pubs.rsc.org/en/content/articlepdf/2024/SC/D3SC06997D

Reference48 articles.

1. Age-Dependence and Aging-Dependence: Neuronal Loss and Lifespan in a C. elegans Model of Parkinson’s Disease

2. Diagnosis and Treatment of Parkinson Disease

3. Effects of deep brain stimulation and medication on bradykinesia and muscle activation in Parkinson's disease

4. Beyond tremor and rigidity: non-motor features of Parkinson’s disease

5. Motor symptoms in Parkinson’s disease: A unified framework

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