Dihydropyrazine suppresses TLR4-dependent inflammatory responses by blocking MAPK signaling in human hepatoma HepG2 cells
Author:
Affiliation:
1. School of Pharmacy at Fukuoka, International University of Health and Welfare
2. Faculty of Pharmaceutical Sciences, Sojo University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/47/9/47_381/_pdf
Reference32 articles.
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2. Akira, S. and Takeda, K. (2004): Toll-like receptor signalling. Nat. Rev. Immunol., 4, 499-511.
3. Aragno, M. and Mastrocola, R. (2017): Dietary sugars and endogenous formation of advanced glycation endproducts: emerging mechanisms of disease. Nutrients, 9.
4. Del Pino-García, R., González-SanJosé, M.L., Rivero-Pérez, M.D. and Muñiz, P. (2012): Influence of the degree of roasting on the antioxidant capacity and genoprotective effect of instant coffee: contribution of the melanoidin fraction. J. Agric. Food Chem., 60, 10530-10539.
5. Esaki, M., Ishida, T., Miyauchi, Y. and Takechi, S. (2020): The effect of dihydropyrazines on lipopolysaccharide-stimulated human hepatoma HepG2 cells via regulating the TLR4-MyD88-mediated NF-κB signaling pathway. J. Toxicol. Sci., 45, 401-409.
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2. Oxidative stress and cellular toxicity induced by dihydropyrazine: a comparative study with other Maillard reaction products;The Journal of Toxicological Sciences;2023
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