Functional modulation of liver mitochondria in lipopolysaccharide/drug co-treated rat liver injury model
Author:
Affiliation:
1. The Laboratory of Biopharmaceutics, Graduate School of Pharmaceutical Sciences, Chiba University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/44/12/44_833/_pdf
Reference58 articles.
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3. Biasutto, L., Azzolini, M., Szabò, I. and Zoratti, M. (2016): The mitochondrial permeability transition pore in AD 2016: an update. Biochim. Biophys. Acta, 1863, 2515-2530.
4. Boelsterli, U.A. (2003): Diclofenac-induced liver injury: a paradigm of idiosyncratic drug toxicity. Toxicol. Appl. Pharmacol., 192, 307-322.
5. Cai, S.Y., Ouyang, X., Chen, Y., Soroka, C.J., Wang, J., Mennone, A., Wang, Y., Mehal, W.Z., Jain, D. and Boyer, J.L. (2017): Bile acids initiate cholestatic liver injury by triggering a hepatocyte-specific inflammatory response. JCI Insight, 2, e90780.
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