Lipopolysaccharide administration increases the susceptibility of mitochondrial permeability transition pore opening via altering adenine nucleotide translocase conformation in the mouse liver
Author:
Affiliation:
1. Laboratory of Biopharmaceutics, Graduate School of Pharmaceutical Sciences, Chiba University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/48/2/48_65/_pdf
Reference36 articles.
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2. Arakawa, K., Ikeyama, Y., Sato, T., Segawa, M., Sekine, S. and Ito, K. (2019): Functional modulation of liver mitochondria in lipopolysaccharide/drug co-treated rat liver injury model. J. Toxicol. Sci., 44, 833-843.
3. Bertholet, A.M., Chouchani, E.T., Kazak, L., Angelin, A., Fedorenko, A., Long, J.Z., Vidoni, S., Garrity, R., Cho, J., Terada, N., Wallace, D.C., Spiegelman, B.M. and Kirichok, Y. (2019): H+ transport is an integral function of the mitochondrial ADP/ATP carrier. Nature, 571, 515-520.
4. Beyer, K. and Klingenberg, M. (1985): ADP/ATP carrier protein from beef heart mitochondria has high amounts of tightly bound cardiolipin, as revealed by 31P nuclear magnetic resonance. Biochemistry, 24, 3821-3826.
5. Beyer, K. and Nuscher, B. (1996): Specific cardiolipin binding interferes with labeling of sulfhydryl residues in the adenosine diphosphate/adenosine triphosphate carrier protein from beef heart mitochondria. Biochemistry, 35, 15784-15790.
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