Increased susceptibility to troglitazone-induced mitochondrial permeability transition in type 2 diabetes mellitus model rat
Author:
Affiliation:
1. Laboratory of Biopharmaceutics, Graduate School of Pharmaceutical Sciences, Chiba University
2. Department of Investigative Toxicology, Nonclinical Research Center, Tokushima Research Institute, Otsuka Pharmaceutical Co., Ltd.
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/43/5/43_339/_pdf
Reference41 articles.
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2. Bae, M.A., Rhee, H. and Song, B.J. (2003): Troglitazone but not rosiglitazone induces G1 cell cycle arrest and apoptosis in human and rat hepatoma cell lines. Toxicol. Lett., 139, 67-75.
3. Baker, M.A., Cerniglia, G.J. and Zaman, A. (1990): Microtiter plate assay for the measurement of glutathione and glutathione disulfide in large numbers of biological samples. Anal. Biochem., 190, 360-365.
4. Bell, C.C., Hendriks, D.F., Moro, S.M., Ellis, E., Walsh, J., Renblom, A., Fredriksson Puigvert, L., Dankers, A.C., Jacobs, F., Snoeys, J., Sison-Young, R.L., Jenkins, R.E., Nordling, Å., Mkrtchian, S., Park, B.K., Kitteringham, N.R., Goldring, C.E., Lauschke, V.M. and Ingelman-Sundberg, M. (2016): Characterization of primary human hepatocyte spheroids as a model system for drug-induced liver injury, liver function and disease. Sci. Rep., 6, 25187.
5. Bligh, E.G. and Dyer, W.J. (1959): A rapid method of total lipid extraction and purification. Can. J. Biochem. Physiol., 37, 911-917.
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