Fluoride-induced iron overload contributes to hepatic oxidative damage in mouse and the protective role of Grape seed proanthocyanidin extract
Author:
Affiliation:
1. Department of Public Health, Medical College, Shihezi University, China
2. Occupational disease prevention and control of hospital of the Xinjiang Uygur Autonomous Region, China
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/43/5/43_311/_pdf
Reference31 articles.
1. Agalakova, N.I. and Gusev, G.P. (2011): Fluoride-induced death of rat erythrocytes in vitro. Toxicol. In Vitro, 25, 1609-1618.
2. Arezes, J. and Nemeth, E. (2015): Hepcidin and iron disorders: new biology and clinical approaches. Int. J. Lab. Hematol., 37 (Suppl 1), 92-98.
3. Attia, S.M., Al-Bakheet, S.A. and Al-Rasheed, N.M. (2010): Proanthocyanidins produce significant attenuation of doxorubicin-induced mutagenicity via suppression of oxidative stress. Oxid. Med. Cell. Longev., 3, 404-413.
4. Bagchi, D., Swaroop, A., Preuss, H.G. and Bagchi, M. (2014): Free radical scavenging, antioxidant and cancer chemoprevention by grape seed proanthocyanidin: an overview. Mutat. Res., 768, 69-73.
5. Bayeva, M., Chang, H.C., Wu, R. and Ardehali, H. (2013): When less is more: novel mechanisms of iron conservation. Trends Endocrinol. Metab., 24, 569-577.
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