Reversible and monitorable nephrotoxicity in rats by the novel potent transcriptional enhanced associate domain (TEAD) inhibitor, K-975
Author:
Affiliation:
1. Toxicological Research Laboratories, Research Unit, Research Division, Kyowa Kirin Co., Ltd.
2. Laboratory of Molecular Toxicology, School of Pharmaceutical Sciences, University of Shizuoka
Publisher
Japanese Society of Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/49/4/49_175/_pdf
Reference62 articles.
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2. Azzolin, L., Panciera, T., Soligo, S., Enzo, E., Bicciato, S., Dupont, S., Bresolin, S., Frasson, C., Basso, G., Guzzardo, V., Fassina, A., Cordenonsi, M. and Piccolo, S. (2014): YAP/TAZ incorporation in the β-catenin destruction complex orchestrates the Wnt response. Cell, 158, 157-170. Elsevier.
3. Camargo, F.D., Gokhale, S., Johnnidis, J.B., Fu, D., Bell, G.W., Jaenisch, R. and Brummelkamp, T.R. (2007): YAP1 increases organ size and expands undifferentiated progenitor cells. Curr. Biol., 17, 2054-2060.
4. Chan, P., Han, X., Zheng, B., DeRan, M., Yu, J., Jarugumilli, G.K., Deng, H., Pan, D., Luo, X. and Wu, X. (2016): Autopalmitoylation of TEAD proteins regulates transcriptional output of the Hippo pathway. Nat. Chem. Biol., 12, 282-289.
5. Chen, J., Wang, X., He, Q. and Harris, R.C. (2022): TAZ is important for maintenance of the integrity of podocytes. Am. J. Physiol. Renal Physiol., 322, F419-F428.
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