Role of ionotropic glutamatergic receptors and nitric oxide in the effects of flutriafol, a triazole fungicide, on the in vivo striatal dopamine release
Author:
Affiliation:
1. Department of Functional Biology and Helth Sciences, Faculty of Biology, University of Vigo, Spain
2. Institute of Biological Sciences, Federal University of Pará, Brazil
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/37/6/37_1135/_pdf
Reference39 articles.
1. Brenmann, J.E. and Bredt, D.S. (1997): Synaptic signaling by nitric oxide. Curr. Opinion Neurobiol., 7, 374-378.
2. Campos, F., Alfonso, M., Vidal, L., Faro, L.R.F. and Durán, R. (2006): Mediation of glutamatergic receptors and nitric oxide on striatal dopamine release evoked by anatoxin-a. An in vivo microdialysis study. Eur. J. Pharmacol., 548, 90-98.
3. Crofton, K.M. (1996): A structure-activity relationship for the neurotoxicity of triazole fungicides. Toxicol. Lett., 84, 155-159.
4. Crofton, K.M., Boncek, V.M. and Reiter, L.W. (1988): Hyperactivity induced by triadimefom, a triazole fungicide. Fundam. Appl. Toxicol., 10, 459-465.
5. Crofton, K.M., Boncek, V.M. and Macphail, R.C. (1989): Evidence for monoaminergic involvement in triadimefon-induced hyperactivity. Psychopharmacology, 97, 326-330.
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