Direct Involvement of Arachidonic Acid in the Development of Ear Edema via TRPV3
Author:
Affiliation:
1. Shionogi Innovation Center for Drug Discovery, Shionogi & Co., Ltd.
2. Shionogi Pharmaceutical Research Center, Shionogi & Co., Ltd.
3. Faculty of Advanced Life Science, Hokkaido University
Publisher
Japan Oil Chemists' Society
Subject
General Chemical Engineering,General Medicine,General Chemistry
Link
https://www.jstage.jst.go.jp/article/jos/66/6/66_ess16227/_pdf
Reference40 articles.
1. 1) Chapkin, R.S.; Ziboh, V.A.; Marcelo, C.L.; Voorhees, J.J. Metabolism of essential fatty acids by human epidermal enzyme preparations: evidence of chain elongation. J. Lipid Res. 27, 945-954 (1986).
2. 2) Crummey, A.; Harper, G.P.; Boyle, E.A.; Mangan, F.R. Inhibition of arachidonic acid-induced ear oedema as a model for assessing topical anti-inflammatory compounds. Agents Actions 20, 69-76 (1987).
3. 3) Inoue, H.; Mori, T.; Koshihara, Y. Sulfidopeptide-leukotrienes are major mediators of arachidonic acid-induced mouse ear edema. Prostaglandins 36, 731-739 (1988).
4. 4) Nilius, B.; Bíró, T.; Owsianik, G. TRPV3: time to decipher a poorly understood family member! J. Physiol. 592, 295-304 (2014).
5. 5) Peier, A.M.; Reeve, A.J.; Andersson, D.A.; Moqrich, A.; Earley, T.J.; Hergarden, A.C.; Story, G.M.; Colley, S.; Hogenesch, J.B.; McIntyre, P.; Bevan, S.; Patapoutian, A. A heat-sensitive TRP channel expressed in keratinocytes. Science 296, 2046-2049 (2002).
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