Defining the STATus quo in muscle hypertrophy. Focus on “Overload-mediated skeletal muscle hypertrophy is not impaired by loss of myofiber STAT3”
Author:
Affiliation:
1. East Carolina Diabetes and Obesity Institute, Brody School of Medicine, East Carolina University, Greenville, North Carolina; and
2. Department of Physiology, Brody School of Medicine, East Carolina University, Greenville, North Carolina
Publisher
American Physiological Society
Subject
Cell Biology,Physiology
Link
https://www.physiology.org/doi/pdf/10.1152/ajpcell.00165.2017
Reference8 articles.
1. Early Activation of Rat Skeletal Muscle IL-6/STAT1/STAT3 Dependent Gene Expression in Resistance Exercise Linked to Hypertrophy
2. Muscle wasting in disease: molecular mechanisms and promising therapies
3. Overload-mediated skeletal muscle hypertrophy is not impaired by loss of myofiber STAT3
4. Interleukin-6 Is an Essential Regulator of Satellite Cell-Mediated Skeletal Muscle Hypertrophy
5. Inhibition of Stat3 Activation Suppresses Caspase-3 and the Ubiquitin-Proteasome System, Leading to Preservation of Muscle Mass in Cancer Cachexia
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. The JAK/STAT Pathway in Skeletal Muscle Pathophysiology;Frontiers in Physiology;2019-04-30
2. STAT3 in Skeletal Muscle Function and Disorders;International Journal of Molecular Sciences;2018-08-02
3. Overload-mediated skeletal muscle hypertrophy is not impaired by loss of myofiber STAT3;American Journal of Physiology-Cell Physiology;2017-09-01
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