A novel role for sphingolipid metabolism in oxidant-mediated skeletal muscle fatigue. Focus on “Sphingomyelinase stimulates oxidant signaling to weaken skeletal muscle and promote fatigue”-Reference-Cited by-同舟云学术

A novel role for sphingolipid metabolism in oxidant-mediated skeletal muscle fatigue. Focus on “Sphingomyelinase stimulates oxidant signaling to weaken skeletal muscle and promote fatigue”

Published:2010-09 Issue:3 Volume:299 Page:C549-C551
ISSN:0363-6143
Container-title:American Journal of Physiology-Cell Physiology
language:en
Short-container-title:American Journal of Physiology-Cell Physiology

Author:

Cowart L. Ashley¹

Affiliation:

1. Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, and the Ralph H. Johnson Veterans Affairs Medical Center, Charleston, South Carolina

Publisher

American Physiological Society

Subject

Cell Biology,Physiology

Link

https://www.physiology.org/doi/pdf/10.1152/ajpcell.00236.2010

Reference27 articles.

1. Sphingomyelin hydrolysis during apoptosis

2. Endurance training in obese humans improves glucose tolerance and mitochondrial fatty acid oxidation and alters muscle lipid content

3. Pleiotropic effects of sphingolipids in skeletal muscle

4. Characterizing the effects of saturated fatty acids on insulin signaling and ceramide and diacylglycerol accumulation in 3T3-L1 adipocytes and C2C12 myotubes

5. Neutral sphingomyelinases and nSMase2: Bridging the gaps

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