B2 cells contribute to hypertension and natural killer cell activation possibly via AT1-AA in response to placental ischemia-Reference-Cited by-同舟云学术

B2 cells contribute to hypertension and natural killer cell activation possibly via AT1-AA in response to placental ischemia

Published:2023-02-01 Issue:2 Volume:324 Page:F179-F192
ISSN:1931-857X
Container-title:American Journal of Physiology-Renal Physiology
language:en
Short-container-title:American Journal of Physiology-Renal Physiology

Author:

Herrock Owen T.¹^ORCID,Deer Evangeline¹^ORCID,Amaral Lorena M.¹^ORCID,Campbell Nathan¹,Lemon James¹,Ingram Nicole¹,Cornelius Denise C.²^ORCID,Turner Ty W.¹,Fitzgerald Sarah¹,Ibrahim Tarek¹,Dechend Ralf³,Wallukat Gerd³,LaMarca Babbette¹⁴^ORCID

Affiliation:

1. Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, Mississippi

2. Emergency Medicine, University of Mississippi Medical Center, Jackson, Mississippi

3. Experimental and Clinical Research Center, HELIOS Clinic, Max-Delbrück-Centrum für Molekulare Medizin, Berlin, Germany

4. Department of Obstetrics and Gynecology, University of Mississippi Medical Center, Jackson, Mississippi

Abstract

This study demonstrates that placental ischemia-stimulated B2 cells induce hypertension and circulating natural killer cell activation and angiotensin II type 1 receptor production in normal pregnant rats.

Funder

HHS | NIH | Eunice Kennedy Shriver National Institute of Child Health and Human Development

HHS | NIH | National Institute of General Medical Sciences

American Heart Association

HHS | NIH | National Heart, Lung, and Blood Institute

Publisher

American Physiological Society

Subject

Physiology

Link

https://journals.physiology.org/doi/pdf/10.1152/ajprenal.00190.2022

Reference88 articles.