Multiple Interactions of the Oct-1 (POU2F1) Transcription Factor with PORE and MORE Sites
Author:
Publisher
Pleiades Publishing Ltd
Subject
Structural Biology,Biophysics
Link
http://link.springer.com/content/pdf/10.1134/S0026893319030191.pdf
Reference19 articles.
1. Kang J., Gemberling M., Nakamura M., Whitby F.G., Handa H., Fairbrother W.G., Tantin D. 2009. A general mechanism for transcription regulation by Oct1 and Oct4 in response to genotoxic and oxidative stress. Genes Dev. 23, 208–222.
2. Xiao S., Liao S., Zhou Y., Jang B., Li Y., Xue M. 2014. High expression of octamer transcription factor 1 in cervical cancer. Oncol. Lett. 7, 1889–1894.
3. Maddox J., Shakya A., South S., Shelton D., Andersen J.N., Chidester S., Kang J., Gligorich K.M., Jones D., Spangrude G.J., Welm B.E., Tantin D. 2012. Transcription factor Oct1 is a somatic and cancer stem cell determinant. PLoS Genet. 8, e1003048. https://doi.org/10.1371/journal.pgen.1003048
4. Wang Y.P., Song G.H., Chen J., Xiao C., Li C., Zhong L., Sun X., Wang Z.W., Deng G.L., Yu F.D., Xue Y.M., Tang H.M., Peng Z.H., Wang X.L. 2016. Elevated Oct-1 participates in colon tumorigenesis and independently predicts poor prognoses of colorectal cancer patients. Tumor Biol. 37, 3247–3255.
5. Pankratova E.V., Stepchenko A.G., Krylova I.D., Portseva T.N., Georgieva S.G. 2018. The regulatory interplay between Oct-1 isoforms contributes to hematopoiesis and the isoforms imbalance correlates with a malignant transformation of B cells. Oncotarget. 9, 29892–29905.
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Suppression of OCT-1 in Metastatic Breast Cancer Cells Reduces Tumor Metastatic Potential, Hypoxia Resistance, and Drug Resistance;Life;2022-09-15
2. Reduced Expression of the Tissue-Specific Oct-1L Isoform Exerts an Antitumor Effect on Namalwa Burkitt’s Lymphoma Cells;Molecular Biology;2022-08
3. POU2F1 (Oct-1) Differently Autoregulates the Alternative Promoters of Its Own Gene by Binding to Different Regulatory Sites;Molecular Biology;2021-11
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3