Role of Nitric Oxide Synthase in the Infarct-Limiting Effect of Normobaric Hypoxia
Author:
Publisher
Pleiades Publishing Ltd
Subject
Physiology,Biochemistry,Ecology, Evolution, Behavior and Systematics
Link
https://link.springer.com/content/pdf/10.1134/S0022093022040202.pdf
Reference22 articles.
1. Baxter GF, Ferdinandy P (2001) Delayed preconditioning of myocardium: current perspectives. Basic Res Cardiol 96: 329–344. https://doi.org/10.1007/s003950170041
2. Tsibulnikov SY, Maslov LN, Naryzhnaya NV, Ma H, Lishmanov YB, Oeltgen PR, Garlid K (2018) Role of protein kinase C, PI3 kinase, tyrosine kinases, NO-synthase, KATP channels and MPT pore in the signaling pathway of the cardioprotective effect of chronic continuous hypoxia. Gen Physiol Biophys 37: 537–547. https://doi.org/10.4149/gpb_2018013
3. Guo Y, Stein AB, Wu WJ, Zhu X, Tan W, Li Q, Bolli R (2005) Late preconditioning induced by NO donors, adenosine A1 receptor agonists, and δ1-opioid receptor agonists is mediated by iNOS. Am J Physiol Heart Circ Physiol 289: H22517. https://doi.org/10.1152/ajpheart.00341.2005
4. Maslov LN, Naryzhnaya NV, Tsibulnikov SYu, Kolar F, Zhang Y, Wang H, Gusakova AM, Lishmanov YuB (2013) Role of endogenous opioid peptides in the infarct size-limiting effect of adaptation to chronic continuous hypoxia. Life Sci 93: 373–379. https://doi.org/10.1016/j.lfs.2013.07.018
5. Chiari PC, Bienengraeber MW, Weihrauch D, Krolikowski JG, Kersten JR, Warltier DC, Pagel PS (2005) Role of endothelial nitric oxide synthase as a trigger and mediator of isoflurane-induced delayed preconditioning in rabbit myocardium. Anesthesiology 103: 74–83. https://doi.org/10.1097/00000542-200507000-00014
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