4. Entero-pancreatic Endocrine Function and Diabetes
Author:
Affiliation:
1. Department of Diabetes, Endocrinology and Nutrition, Graduate School of Medicine, Kyoto University
Publisher
Japanese Society of Internal Medicine
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/naika/108/9/108_1713/_pdf
Reference17 articles.
1. 1) Inagaki N, et al: Reconstitution of IKATP: an inward rectifier subunit plus the sulfonylurea receptor. Science 270: 1166-1170, 1995.
2. 2) Inagaki N, et al: A family of sulfonylurea receptors determines the pharmacological properties of ATP-sensitive K+ channels. Neuron 16: 1011-1017, 1996.
3. 3) Yorifuji T, et al: The C42R mutation in the Kir6.2 (KCNJ11) gene as a cause of transient neonatal diabetes, childhood diabetes, or later-onset, apparently type 2 diabetes mellitus. J Clin Endocrinol Metab 90: 3174-3178, 2005.
4. 4) Pearson ER, et al: Switching from insulin to oral sulfonylureas in patients with diabetes due to Kir6.2 mutations. N Engl J Med 355: 467-477, 2006.
5. 5) Nagashima K, et al: Epidemiology, clinical characteristics, and genetic etiology of neonatal diabetes in Japan. Pediatr Int 59: 129-133, 2017.
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