The C42R Mutation in the Kir6.2 (KCNJ11) Gene as a Cause of Transient Neonatal Diabetes, Childhood Diabetes, or Later-Onset, Apparently Type 2 Diabetes Mellitus-Reference-Cited by-同舟云学术

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The C42R Mutation in the Kir6.2 (KCNJ11) Gene as a Cause of Transient Neonatal Diabetes, Childhood Diabetes, or Later-Onset, Apparently Type 2 Diabetes Mellitus

Published:2005-06 Issue:6 Volume:90 Page:3174-3178
ISSN:0021-972X
Container-title:The Journal of Clinical Endocrinology & Metabolism
language:en
Short-container-title:The Journal of Clinical Endocrinology & Metabolism

Author:

Yorifuji Tohru,Nagashima Kazuaki,Kurokawa Keiji,Kawai Masahiko,Oishi Mariko,Akazawa Yoshiharu,Hosokawa Masaya,Yamada Yuichiro,Inagaki Nobuya,Nakahata Tatsutoshi

Publisher

The Endocrine Society

Subject

Biochemistry, medical,Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Cited by 107 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults;eLife;2024-05-03

2. The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults;2024-04-15

3. A synonymous KCNJ11 variant leading to MODY13: A case report and literature review;Molecular Genetics and Metabolism Reports;2024-03

4. Monogenic Causes of Diabetes;Textbook of Diabetes;2024-01-12

5. A Japanese school urine screening program led to the diagnosis of KCNJ11-MODY: A case report;CLIN PEDIATR ENDOCRI;2024

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