Comparative airway inflammatory response of normal volunteers to ozone and lipopolysaccharide challenge-Reference-Cited by-同舟云学术

Comparative airway inflammatory response of normal volunteers to ozone and lipopolysaccharide challenge

Published:2010-06-11 Issue:8 Volume:22 Page:648-656
ISSN:0895-8378
Container-title:Inhalation Toxicology
language:en
Short-container-title:Inhalation Toxicology

Author:

Hernandez Michelle L.,Harris Bradford,Lay John C.,Bromberg Philip A.,Diaz-Sanchez David,Devlin Robert B.,Kleeberger Steven R.,Alexis Neil E.,Peden David B.

Publisher

Informa UK Limited

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

http://www.tandfonline.com/doi/pdf/10.3109/08958371003610966

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1. Blunting airway eosinophilic inflammation results in a decreased airway neutrophil response to inhaled LPS in patients with atopic asthma: A role for CD14

2. CD14-dependent airway neutrophil response to inhaled LPS: Role of atopy

3. Effect of inhaled endotoxin on airway and circulating inflammatory cell phagocytosis and CD11b expression in atopic asthmatic subjects

4. Inhalation of low-dose endotoxin favors local TH2 response and primes airway phagocytes in vivo

5. Acute LPS inhalation in healthy volunteers induces dendritic cell maturation in vivo

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