ER stress and the decline and fall of pancreatic beta cells in type 1 diabetes
Author:
Funder
Juvenile Diabetes Research Foundation
Publisher
Uppsala Medical Society
Subject
General Medicine
Link
http://www.tandfonline.com/doi/pdf/10.3109/03009734.2015.1135217
Reference71 articles.
1. Glucose stimulates proinsulin biosynthesis by a dose-dependent recruitment of pancreatic beta cells.
2. Mutant analysis links the translocon and BiP to retrograde protein transport for ER degradation
3. The Requirement for Molecular Chaperones during Endoplasmic Reticulum-associated Protein Degradation Demonstrates That Protein Export and Import Are Mechanistically Distinct
4. Lectin chaperones help direct the maturation of glycoproteins in the endoplasmic reticulum
5. The IRE1α/XBP1s Pathway Is Essential for the Glucose Response and Protection of β Cells
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