State of the field: An informatics-based systematic review of the SOD1-G93A amyotrophic lateral sclerosis transgenic mouse model
Author:
Publisher
Informa UK Limited
Subject
Neurology (clinical),Neurology
Link
http://www.tandfonline.com/doi/pdf/10.3109/21678421.2015.1047455
Reference87 articles.
1. Amyotrophic lateral sclerosis
2. Characterization of the Contribution of Genetic Background and Gender to Disease Progression in the SOD1 G93A Mouse Model of Amyotrophic Lateral Sclerosis: A Meta-Analysis
3. Alteration of the Bcl-x/Bax Ratio in a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis: Evidence for the Implication of the p53 Signaling Pathway
4. Cargo distributions differentiate pathological axonal transport impairments
5. Dysregulation of iron protein expression in the G93A model of amyotrophic lateral sclerosis
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1. Mass spectrometry imaging of SOD1 protein-metal complexes in SOD1G93A transgenic mice implicates demetalation with pathology;Nature Communications;2024-08-08
2. Montelukast improves disease outcome in SOD1G93A female mice by counteracting oligodendrocyte dysfunction and aberrant glial reactivity;British Journal of Pharmacology;2024-05-15
3. Repeated mild traumatic brain injury triggers pathology in asymptomatic C9ORF72 transgenic mice;Brain;2023-08-01
4. Comparing therapeutic modulators of the SOD1 G93A Amyotrophic Lateral Sclerosis mouse pathophysiology;Frontiers in Neuroscience;2023-01-19
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