Alteration of the Bcl-x/Bax Ratio in a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis: Evidence for the Implication of the p53 Signaling Pathway
Author:
Publisher
Elsevier BV
Subject
Neurology
Reference40 articles.
1. p53 is essential for developmental neuron death as regulated by the TrkA and p75 neurotrophin receptors;Aloyz;J. Cell Biol.,1998
2. Gene regulation analysis by lipopolyamine-mediated DNA transfer in primary neurons;Barthel;Methods Neurosci.,1992
3. Bcl-xL protects adult septal cholinergic neurons from axotomized cell death;Blomer;Proc. Natl. Acad. Sci. USA,1998
4. Bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death;Boise;Cell,1993
5. ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions;Bruijn;Neuron,1997
Cited by 97 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Redox dysregulation as a driver for DNA damage and its relationship to neurodegenerative diseases;Translational Neurodegeneration;2023-04-14
2. The paradigm of amyloid precursor protein in amyotrophic lateral sclerosis: The potential role of the 682YENPTY687 motif;Computational and Structural Biotechnology Journal;2023
3. Catching a killer: Mechanisms of programmed cell death and immune activation in Amyotrophic Lateral Sclerosis;Immunological Reviews;2022-05-07
4. The Complex Mechanisms by Which Neurons Die Following DNA Damage in Neurodegenerative Diseases;International Journal of Molecular Sciences;2022-02-24
5. Regulated cell death: discovery, features and implications for neurodegenerative diseases;Cell Communication and Signaling;2021-12
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3