Specific restriction of cholesterol from cortical lens gap junctional membrane in the U18666A cataract
Author:
Publisher
Informa UK Limited
Subject
Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology
Link
http://www.tandfonline.com/doi/pdf/10.3109/02713688809015150
Reference19 articles.
1. Source of cholesterol for the ocular lens, studied with U18666A: A cataract-producing inhibitor of lipid metabolism
2. Regional distribution of lipids and phospholipase A2activity in normal and cataractous rat lens
3. Limited proteolysis of MP26 in lens fiber plasma membranes of the U18666A-induced cataract in rats
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1. Cellular mechanism of U18666A-mediated apoptosis in cultured murine cortical neurons: Bridging Niemann–Pick disease type C and Alzheimer's disease;Cellular Signalling;2006-11
2. Sphingolipids in human lens membranes: an update on their composition and possible biological implications;Chemistry and Physics of Lipids;2004-04
3. Cholesterol and cataracts;Survey of Ophthalmology;1996-01
4. Plasma membrane-cytoskeleton complex in the normal and cataractous lens;Cytoskeleton in Specialized Tissues and in Pathological States;1996
5. Calcium Activated Proteolysis and Protein Modification in the U18666A Cataract;Experimental Eye Research;1993-12
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