Limited proteolysis of MP26 in lens fiber plasma membranes of the U18666A-induced cataract in rats
Author:
Publisher
Informa UK Limited
Subject
Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology
Link
http://www.tandfonline.com/doi/pdf/10.3109/02713689509000008
Reference19 articles.
1. Inhibition of Cholesterol Biosynthesis in the Rat by 3 -(2-diethylaminoethoxy)androst-5-en-17-one, hydrochloride
2. Mechanism of cataract production by 3-β(2-diethylaminoethoxy) androst-5-en-17-one hydrochloride, U18666A: An inhibitor of cholesterol biosynthesis
3. Source of cholesterol for the ocular lens, studied with U18666A: A cataract-producing inhibitor of lipid metabolism
4. Composition and synthesis in vivo of water-insoluble proteins in the rat ocular lens: Normal and cataractous
5. Protein composition of bovine lens cortical fiber cell membranes
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1. Cellular mechanism of U18666A-mediated apoptosis in cultured murine cortical neurons: Bridging Niemann–Pick disease type C and Alzheimer's disease;Cellular Signalling;2006-11
2. The Toxicology of HMG—CoA Reductase Inhibitors: Prediction of Human Risk;Toxicologic Pathology;2004-02
3. Evidence for Distinct Cholesterol Domains in Fiber Cell Membranes from Cataractous Human Lenses;Journal of Biological Chemistry;2001-04
4. Hyperbaric Oxygen in vivo Accelerates the Loss of Cytoskeletal Proteins and MIP26 in Guinea Pig Lens Nucleus;Experimental Eye Research;1999-04
5. Comparison of the Water Transporting Properties of MIP and AQP1;The Journal of Membrane Biology;1997-09
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