Effect of phosphatase and tensin homology deleted on chromosome 10 (PTEN) gene transfection on reversal of multidrug resistance in K562/ADM cells
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
http://www.tandfonline.com/doi/pdf/10.3109/10428194.2011.650695
Reference22 articles.
1. Reversal of P-Glycoprotein-mediated Paclitaxel Resistance by New Synthetic Isoprenoids in Human Bladder Cancer Cell Line
2. Expression of Drug Resistance Proteins Pgp, MRP1, MRP3, MRP5 AND GST-π in Human Glioma
3. P-TEN, the tumor suppressor from human chromosome 10q23, is a dual-specificity phosphatase
4. Survival of acute myeloid leukemia cells requires PI3 kinase activation
5. PTEN is inversely correlated with the cell survival factor Akt/PKB and is inactivated via multiple mechanismsin haematological malignancies
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2. TRIB2 knockdown as a regulator of chemotherapy resistance and proliferation via the ERK/STAT3 signaling pathway in human chronic myelogenous leukemia K562/ADM cells;Oncology Reports;2018-02-07
3. Improving nelarabine efficacy in T cell acute lymphoblastic leukemia by targeting aberrant PI3K/AKT/mTOR signaling pathway;Journal of Hematology & Oncology;2016-10-24
4. SET protein overexpression contributes to paclitaxel resistance in MCF-7/S cells through PI3K/Akt pathway;Journal of Drug Targeting;2016-10-20
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