SET protein overexpression contributes to paclitaxel resistance in MCF-7/S cells through PI3K/Akt pathway
Author:
Affiliation:
1. Department of Pharmacy, The First Affiliated Hospital of Xi’an Jiaotong University, Xi’an, PR China;
2. Department of Pharmacy, The Eighth Hospital of Xi’an, Xi’an, PR China;
3. School of Pharmacy, Xi’an Jiaotong University, Xi’an, PR China
Funder
National Natural Science Foundation of China
Publisher
Informa UK Limited
Subject
Pharmaceutical Science
Link
https://www.tandfonline.com/doi/pdf/10.1080/1061186X.2016.1245307
Reference28 articles.
1. Metformin reverses multidrug resistance and epithelial–mesenchymal transition (EMT) via activating AMP-activated protein kinase (AMPK) in human breast cancer cells
2. Paclitaxel resistance is associated with switch from apoptotic to autophagic cell death in MCF-7 breast cancer cells
3. A leukotriene B4 receptor-2 is associated with paclitaxel resistance in MCF-7/DOX breast cancer cells
4. Knockdown of cyclophilin A reverses paclitaxel resistance in human endometrial cancer cells via suppression of MAPK kinase pathways
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