Gap-junction coupling and ATP-sensitive potassium channels in humanβ-cell clusters: Effects on emergent dynamics
Author:
Funder
Università degli Studi di Padova
Seventh Framework Programme
Publisher
American Physical Society (APS)
Link
http://harvest.aps.org/v2/journals/articles/10.1103/PhysRevE.96.032403/fulltext
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1. Widespread synchronous [Ca2+]i oscillations due to bursting electrical activity in single pancreatic islets
2. Influence of cell number on the characteristics and synchrony of Ca 2+ oscillations in clusters of mouse pancreatic islet cells
3. Microfluidic glucose stimulation reveals limited coordination of intracellular Ca2+ activity oscillations in pancreatic islets
4. Loss of Connexin36 Channels Alters β-Cell Coupling, Islet Synchronization of Glucose-Induced Ca2+ and Insulin Oscillations, and Basal Insulin Release
5. Cx36-Mediated Coupling Reduces β-Cell Heterogeneity, Confines the Stimulating Glucose Concentration Range, and Affects Insulin Release Kinetics
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