Peptidomimetic Escape Mechanisms Arise via Genetic Diversity in the Ligand-Binding Site of the Hepatitis C Virus NS3/4A Serine Protease-Reference-Cited by-同舟云学术

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Peptidomimetic Escape Mechanisms Arise via Genetic Diversity in the Ligand-Binding Site of the Hepatitis C Virus NS3/4A Serine Protease

Published:2012-03 Issue:3 Volume:142 Page:654-663
ISSN:0016-5085
Container-title:Gastroenterology
language:en
Short-container-title:Gastroenterology

Author:

Welsch Christoph,Shimakami Tetsuro,Hartmann Christoph,Yang Yan,Domingues Francisco S.,Lengauer Thomas,Zeuzem Stefan,Lemon Stanley M.

Publisher

Elsevier BV

Subject

Gastroenterology,Hepatology

Reference28 articles.

1. Peginterferon alfa-2a plus ribavirin for chronic hepatitis C virus infection;Fried;N Engl J Med,2002

2. Peginterferon alfa-2b plus ribavirin compared with interferon alfa-2b plus ribavirin for initial treatment of chronic hepatitis C: a randomised trial;Manns;Lancet,2001

3. Peginterferon alfa-2a and ribavirin for 16 or 24 weeks in HCV genotype 2 or 3;Shiffman;N Engl J Med,2007

4. SCH 503034, a mechanism-based inhibitor of hepatitis C virus NS3 protease, suppresses polyprotein maturation and enhances the antiviral activity of alpha interferon in replicon cells;Malcolm;Antimicrob Agents Chemother,2006

5. Discovery of the HCV NS3/4A protease inhibitor (1R,5S)-N-[3-amino-1-(cyclobutylmethyl)-2,3-dioxopropyl]-3-[2(S)-[[[(1,1-dimethylethyl)amino]carbonyl]amino]-3,3-dimethyl-1-oxobutyl]-6,6-dimethyl-3-azabicyclo[3.1.0]hexan-2(S)-carboxamide (Sch 503034) II;Prongay;J Med Chem,2007

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1. Regulatory Role of Phospholipids in Hepatitis C Virus Replication and Protein Function;Pathogens;2022-01-15

2. Epistatic interactions promote persistence of NS3-Q80K in HCV infection by compensating for protein folding instability;Journal of Biological Chemistry;2021-09

3. Protease Inhibitor Resistance;Handbook of Antimicrobial Resistance;2017

4. Hepatitis C Virus Genotype 1 to 6 Protease Inhibitor Escape Variants: In Vitro Selection, Fitness, and Resistance Patterns in the Context of the Infectious Viral Life Cycle;Antimicrobial Agents and Chemotherapy;2016-06

5. Natural HCV variants with increased replicative fitness due to NS3 helicase mutations in the C-terminal helix α18;Scientific Reports;2016-01-20

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