Abnormal secretion and function of recombinant human factor VII as the result of modification to a calcium binding site caused by a 15-base pair insertion in the F7 gene
Author:
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Cited by 14 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Contribution of pioneer women hematologists in India in the 20th century;Journal of Hematology and Allied Sciences;2022-06-28
2. A novel missense mutation close to the charge-stabilizing system in a patient with congenital factor VII deficiency;Blood Coagulation & Fibrinolysis;2011-06
3. Recombinant coagulation factor VIIa – from molecular to clinical aspects of a versatile haemostatic agent;Thrombosis Research;2010-06
4. More than one intracellular processing bottleneck delays the secretion of coagulation factor VII;Thrombosis and Haemostasis;2008
5. All post-translational modifications except propeptide cleavage are required for optimal secretion of coagulation factor VII;Thrombosis and Haemostasis;2007
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