Prenatal origin of GATA1 mutations may be an initiating step in the development of megakaryocytic leukemia in Down syndrome-Reference-Cited by-同舟云学术

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Prenatal origin of GATA1 mutations may be an initiating step in the development of megakaryocytic leukemia in Down syndrome

Published:2004-09-01 Issue:5 Volume:104 Page:1588-1589
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Taub Jeffrey W.,Mundschau Gina,Ge Yubin,Poulik Janet M.,Qureshi Faisal,Jensen Tanya,James S. Jill,Matherly Larry H.,Wechsler Joshua,Crispino John D.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/104/5/1588/1701409/zh801704001578g.pdf

Reference9 articles.

1. Gurbuxani S, Vyas P, Crispino JD. Recent insights into the mechanisms of myeloid leukemogenesis in Down syndrome. Blood. 2004;103: 399-406.

2. Zipursky A. Transient leukaemia—a benign form of leukaemia in newborn infants with trisomy 21. Br J Haematol.2003;120: 930-938.

3. Wechsler J, Greene M, McDevitt MA, et al. Acquired mutations in GATA1 in the megakaryoblastic leukemia of Down syndrome. Nat Genet.2002;32: 148-152.

4. Finette BA, Rood B, Poseno T, Vacek P, Pueschel S, Homans AC. Atypical background somatic mutant frequencies at the HPRT locus in children and adults with Down syndrome. Mutat Res.1998;403: 35-43.

5. Bao L, Vlcek C, Paces V, Kraus JP. Identification and tissue distribution of human cystathionine β-synthase mRNA isoforms. Arch Biochem Biophys.1998;350: 95-103.

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