Acquired mutations in GATA1 in the megakaryoblastic leukemia of Down syndrome-Reference-Cited by-同舟云学术

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Acquired mutations in GATA1 in the megakaryoblastic leukemia of Down syndrome

Published:2002-08-12 Issue:1 Volume:32 Page:148-152
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Wechsler Joshua,Greene Marianne,McDevitt Michael A.,Anastasi John,Karp Judith E.,Le Beau Michelle M.,Crispino John D.

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng955z.pdf

Reference30 articles.

1. Lange, B. The management of neoplastic disorders of haematopoiesis in children with Down's syndrome. Br. J. Haematol. 110, 512–524 (2000).

2. Dickstein, J.I., Davis, E.M. & Roulston, D. Localization of the chromosome 22 breakpoints in two cases of acute megakaryoblastic leukemia with t(1;22)(p13;q13). Cancer Genet. Cytogenet. 129, 150–154 (2001).

3. Ma, Z. et al. Fusion of two novel genes, RBM15 and MKL1, in the t(1;22)(p13;q13) of acute megakaryoblastic leukemia. Nature Genet. 28, 220–221 (2001).

4. Tsang, A.P. et al. FOG, a multitype zinc finger protein, acts as a cofactor for transcription factor GATA-1 in erythroid and megakaryocytic differentiation. Cell 90, 109–119 (1997).

5. Visvader, J.E., Crossley, M., Hill, J., Orkin, S.H. & Adams, J.M. The C-terminal zinc finger of GATA-1 or GATA-2 is sufficient to induce megakaryocytic differentiation of an early myeloid cell line. Mol. Cell. Biol. 15, 634–641 (1995).

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