In the absence of IGF-1 signaling, IFN-γ suppresses human malignant T-cell growth
Author:
Affiliation:
1. Center for Experimental Research and Medical Studies (CERMS), San Giovanni Battista Hospital, Turin, Italy;
2. Department of Medicine and Experimental Oncology, Section of Pathology, University of Turin, Italy
Abstract
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/109/6/2496/1477747/zh800607002496.pdf
Reference62 articles.
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2. Pestka S, Kotenko SV, Muthukumaran G, Izotova LS, Cook JR, Garotta G. The interferon gamma (IFN-gamma) receptor: a paradigm for the multichain cytokine receptor. Cytokine Growth Factor Rev1997; 8:189–206.
3. Kaplan DH, Greenlund AC, Tanner JW, Shaw AS, Schreiber RD. Identification of an interferon-gamma receptor alpha chain sequence required for JAK-1 binding. J Biol Chem1996; 271:9–12.
4. Watling D, Guschin D, Muller M, et al. Complementation by the protein tyrosine kinase JAK2 of a mutant cell line defective in the interferon-gamma signal transduction pathway. Nature1993; 366:166–170.
5. Sakatsume M, Igarashi K, Winestock KD, Garotta G, Larner AC, Finbloom DS. The Jak kinases differentially associate with the alpha and beta (accessory factor) chains of the interferon gamma receptor to form a functional receptor unit capable of activating STAT transcription factors. J Biol Chem1995; 270:17528–17534.
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