Effects of the JAK2 mutation on the hematopoietic stem and progenitor compartment in human myeloproliferative neoplasms
Author:
Affiliation:
1. Department of Haematology, University of Cambridge, Cambridge Institute for Medical Research, Cambridge, United Kingdom; and
2. Cambridge University Foundation Hospital Trust, Cambridge, United Kingdom
Abstract
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/118/1/177/1339806/zh802711000177.pdf
Reference20 articles.
1. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders.;Baxter;Lancet,2005
2. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera.;James;Nature,2005
3. A gain-of-function mutation of JAK2 in myeloproliferative disorders.;Kralovics;N Engl J Med,2005
4. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis.;Levine;Cancer Cell,2005
5. JAK2 exon 12 mutations in polycythemia vera and idiopathic erythrocytosis.;Scott;N Engl J Med,2007
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