Targeting BCR-ABL and JAK2 in Ph+ ALL
Author:
Affiliation:
1. SWISS INSTITUTE FOR EXPERIMENTAL CANCER RESEARCH (ISREC)
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/125/9/1362/1388297/1362.pdf
Reference9 articles.
1. Janus kinase inhibition by ruxolitinib extends dasatinib- and dexamethasone-induced remissions in a mouse model of Ph+ ALL.;Appelmann;Blood,2015
2. UKALLXII/ECOG2993: addition of imatinib to a standard treatment regimen enhances long-term outcomes in Philadelphia positive acute lymphoblastic leukemia.;Fielding;Blood,2014
3. Ruxolitinib.;Mesa;Nat Rev Drug Discov,2012
4. JAK of all trades: JAK2-STAT5 as novel therapeutic targets in BCR-ABL1+ chronic myeloid leukemia.;Warsch;Blood,2013
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1. Targeting integrated stress response with ISRIB combined with imatinib treatment attenuates RAS/RAF/MAPK and STAT5 signaling and eradicates chronic myeloid leukemia cells;BMC Cancer;2022-12-02
2. Targeting Integrated Stress Response by ISRIB combined with imatinib attenuates STAT5 signaling and eradicates therapy-resistant Chronic Myeloid Leukemia cells;2021-05-07
3. E2A/PBX1, MLL/AF4, BCR/ABL (M-BCR), BCR/ABL(m-BCR) Gene Rearrangements in Acute Lymphoblastic Leukemia in Iranian Children;Iranian Journal of Pediatrics;2018-05-29
4. Polymeric immunoglobulin receptor promotes tumor growth in hepatocellular carcinoma;Hepatology;2017-04-28
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