Abstract
VEGF exhibits broad vascular functions by activation of its VEGFRs in endothelial cells. In this issue of Blood, Suehiro and colleagues show that VEGF-induced endothelial activities are mediated by activation of Egr-3 transcription factor and that Egr-3 is a potential therapeutic target for angiogenesis-related diseases.
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Cited by
5 articles.
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