Severe anemia in malaria: defense gone wrong?
Author:
Affiliation:
1. KAROLINSKA INSTITUTET
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/106/10/3337/1636424/zh802205003331h.pdf
Reference4 articles.
1. Jakeman GN, Saul A, Hogarth WL, Collins WE. Anaemia of acute malaria infections in non-immune patients primarily results from destruction of uninfected erythrocytes. Parasitology. 1999;119: 127-133.
2. Chang KH, Stevenson MM. Malarial anaemia: mechanisms and implications of insufficient erythropoiesis during blood-stage malaria. Int J Parasitol. 2004;34: 1501-1516.
3. Douki JB, Sterkers Y, Lepolard C, et al. Adhesion of normal and Plasmodium falciparum ring-infected erythrocytes to endothelial cells and the placenta involves the rhoptry-derived ring surface protein-2. Blood. 2003;101: 5025-5032.
4. Waitumbi JN, Donvito B, Kisserli A, Cohen JHM, Soute JA. Age-related changes in red blood cell complement regulatory proteins and susceptibility to severe malaria. J Infect Dis. 2004;190: 1183-1191.
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