Amyloid-β, Tau Protein, and Oxidative Changes as a Physiological Compensatory Mechanism to Maintain CNS Plasticity under Alzheimer's Disease and Other Neurodegenerative Conditions-Reference-Cited by-同舟云学术

Amyloid-β, Tau Protein, and Oxidative Changes as a Physiological Compensatory Mechanism to Maintain CNS Plasticity under Alzheimer's Disease and Other Neurodegenerative Conditions

Published:2009-08-26 Issue:2 Volume:18 Page:381-400
ISSN:1875-8908
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Koudinov Alexei¹²³⁴,Kezlya Elena⁵,Koudinova Natalia¹²³,Berezov Temirbolat¹²³⁴

Affiliation:

1. Berezov Research Laboratory, Russian Academy of Medical Sciences, Moscow, Russia

2. Orechovich Institute of Biomedical Chemistry, RAMS, Moscow, Russia

3. Neurobiology of Lipids, http://www.neurobiologyoflipids.org, Moscow, Russia, Rehovot, Israel

4. Department of Biochemistry, Department of Biochemistry, School of Medicine, Peoples' Friendship University of Russia, Moscow, Russian Federation

5. Interhospital Medical Center “Intermedcenter”, Moscow, Russia

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

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