Peripheral Administration of GSK-3β Antisense Oligonucleotide Improves Learning and Memory in SAMP8 and Tg2576 Mouse Models of Alzheimer’s Disease

Author:

Farr Susan A.12,Sandoval Karin E.3,Niehoff Michael L.2,Witt Ken A.3,Kumar Vijaya B.12,Morley John E.24

Affiliation:

1. Research & Development Service, VA Medical Center, St. Louis, Missouri, USA

2. Division of Geriatric Medicine, Saint Louis University School of Medicine, St. Louis, Missouri, USA

3. Pharmaceutical Sciences, School of Pharmacy, Southern Illinois University Edwardsville, Edwardsville, Illinois, USA

4. Division of Endocrinology, Saint Louis University School of Medicine, St. Louis, Missouri, USA

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Reference45 articles.

1. GSK-3: Functional insights from cell biology and animal models;Kaidanovich-Beilin;Front Mol Neurosci,2011

2. The GSK3 hypothesis of Alzheimer’s disease;Hooper;J Neurochem,2008

3. Glycogen synthase kinase 3 inhibitors in the next horizon for Alzheimer’s disease treatment;Martinez;Int J Alzheimers Dis,2011

4. Baicalin attenuates high fat diet-induced insulin resistance and ectopic fat storage in skeletal muscle, through modulating the protein kinase B/Glycogen synthase kinase 3 beta pathway;Xi;Chin J Nat Med,2016

5. Glycogen synthase kinase-3: A potential preventive target for prostate cancer management;Li;Urol Oncol,2015

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